Search | OpenScholar

Search results

1766 results found

Cysteine 647 in the insulin receptor is required for normal covalent interaction between alpha- and beta-subunits and signal transduction

Publication: Journal Article

Cheatham, and Kahn. 1992. “Cysteine 647 in the Insulin Receptor Is Required for Normal Covalent Interaction Between Alpha- and Beta-Subunits and Signal Transduction”. J Biol Chem 267 (10): 7108-15.

Approaches to the molecular cloning of protein-tyrosine phosphatases in insulin-sensitive tissues

Publication: Journal Article

Goldstein, Zhang, Hashimoto, and Kahn. 1992. “Approaches to the Molecular Cloning of Protein-Tyrosine Phosphatases in Insulin-Sensitive Tissues”. Mol Cell Biochem 109 (2): 107-13.

Insulin stimulation of phosphatidylinositol 3-kinase activity maps to insulin receptor regions required for endogenous substrate phosphorylation

Publication: Journal Article

Backer, Schroeder, Kahn, Myers, Wilden, Cahill, and White. 1992. “Insulin Stimulation of Phosphatidylinositol 3-Kinase Activity Maps to Insulin Receptor Regions Required for Endogenous Substrate Phosphorylation”. J Biol Chem 267 (2): 1367-74.

Quantitative dissociation between EGF effects on c-myc and c-fos gene expression, DNA synthesis, and epidermal growth factor receptor tyrosine kinase activity

Publication: Journal Article

Hauguel-DeMouzon, Csermely, Zoppini, and Kahn. 1992. “Quantitative Dissociation Between EGF Effects on C-Myc and C-Fos Gene Expression, DNA Synthesis, and Epidermal Growth Factor Receptor Tyrosine Kinase Activity”. J Cell Physiol 150 (1): 180-7.

Pertussis toxin inhibits autophosphorylation and activation of the insulin receptor kinase

Publication: Journal Article

Müller-Wieland, White, Behnke, Gebhardt, Neumann, Krone, and Kahn. 1991. “Pertussis Toxin Inhibits Autophosphorylation and Activation of the Insulin Receptor Kinase”. Biochem Biophys Res Commun 181 (3): 1479-85.

Vanadate normalizes hyperglycemia in two mouse models of non-insulin-dependent diabetes mellitus

Publication: Journal Article

Meyerovitch, Rothenberg, Shechter, Bonner-Weir, and Kahn. 1991. “Vanadate Normalizes Hyperglycemia in Two Mouse Models of Non-Insulin-Dependent Diabetes Mellitus”. J Clin Invest 87 (4): 1286-94.

Insulin-like growth factor-mediated phosphorylation and protooncogene induction in Madin-Darby canine kidney cells

Publication: Journal Article

Hauguel-DeMouzon, and Kahn. 1991. “Insulin-Like Growth Factor-Mediated Phosphorylation and Protooncogene Induction in Madin-Darby Canine Kidney Cells”. Mol Endocrinol 5 (1): 51-60.

Deletion of a highly conserved tetrapeptide sequence of the proinsulin connecting peptide (C-peptide) inhibits proinsulin to insulin conversion by transfected pituitary corticotroph (AtT20) cells

Publication: Journal Article

Gross, Villa-Komaroff, Kahn, Weir, and Halban. 1989. “Deletion of a Highly Conserved Tetrapeptide Sequence of the Proinsulin Connecting Peptide (C-Peptide) Inhibits Proinsulin to Insulin Conversion by Transfected Pituitary Corticotroph (AtT20) Cells”. J Biol Chem 264 (36): 21486-90.

Differentiation-dependent phosphorylation of a 175,000 molecular weight protein in response to insulin and insulin-like growth factor-I in L6 skeletal muscle cells

Publication: Journal Article

Beguinot, Kahn, Moses, White, and Smith. 1989. “Differentiation-Dependent Phosphorylation of a 175,000 Molecular Weight Protein in Response to Insulin and Insulin-Like Growth Factor-I in L6 Skeletal Muscle Cells”. Endocrinology 125 (3): 1599-605.

Molecular defects in the insulin receptor in patients with leprechaunism and in their parents

Publication: Journal Article

Reddy, Müller-Wieland, Kriauciunas, and Kahn. 1989. “Molecular Defects in the Insulin Receptor in Patients With Leprechaunism and in Their Parents”. J Lab Clin Med 114 (2): 165-70.