Distinct fibroblast subsets drive inflammation and damage in arthritis
Croft, AP, Campos, Jansen, JD Turner, Marshall, Attar, Savary, et al. 2019. “Distinct Fibroblast Subsets Drive Inflammation and Damage in Arthritis”. Nature 570: 246-51.
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Mixed Effects Association of Single Cells Identifies an Expanded Th1-Skewed Cytotoxic Effector CD4+ T Cell Subset in Rheumatoid Arthritis.
Fonseka, Chamith, Deepak A.# Rao, Nikola C. Teslovich, Susan K. Hannes, Kamil Slowikowski, Michael F. Gurish, Laura T. Donlin, et al. 2018. “Mixed Effects Association of Single Cells Identifies an Expanded Th1-Skewed Cytotoxic Effector CD4+ T Cell Subset in Rheumatoid Arthritis.”. Science Translational Medicine 10 (463).
Discovering in vivo cytokine eQTL interactions from a lupus clinical trial
Davenport, Emma Elisabeth, Tiffany Amariuta, Maria Gutierrez-Arcelus, Kamil Slowikowski, Harm-Jan Westra, Yang Luo, Ciyue Shen, et al. 2018. “Discovering in Vivo Cytokine EQTL Interactions from a Lupus Clinical Trial”. Genome Biology 19 (1): 168.
Methods for high-dimensonal analysis of cells dissociated from cyropreserved synovial tissue
Donlin, Laura T., Deepak A. Rao, Kevin Wei, Kamil Slowikowski, Mandy J. McGeachy, Jason D. Turner, Nida Meednu, et al. 2018. “Methods for High-Dimensonal Analysis of Cells Dissociated from Cyropreserved Synovial Tissue”. Arthritis Research & Therapy 20 (1): 139.
Imputation-based analysis of MICA alleles in the susceptibility to ankylosing spondylitis
Cortes, Adrian, Dafna Gladman, Soumya Raychaudhuri, Jing Cui, Lawrie Wheeler, and Matthew A. Brown. 2018. “Imputation-Based Analysis of MICA Alleles in the Susceptibility to Ankylosing Spondylitis”. Annals of the Rheumatic Diseases 77 (11): 1691-92.
The eMERGE genotype set of 83,717 subjects imputed to ~40 million variants genome wide and association with the herpes zoster medical record phenotype
Stanaway, Ian B., Taryn O. Hall, Elisabeth A. Rosenthal, Melody Palmer, Vivek Naranbhai, Rachel Knevel, Bahram Namjou‐Khales, et al. 2019. “The EMERGE Genotype Set of 83,717 Subjects Imputed to ~40 million Variants Genome Wide and Association With the Herpes Zoster Medical Record Phenotype”. Genetic Epidemiology 43 (1): 63-81.
Lymphocyte innateness is defined by underlying transcriptional states reflecting a balance between proliferation and effector functions
Gutierrez-Arcelus, Maria, Nikola C. Teslovich, Alex Mola, R. B. Polidoro, A. Nathan, H. Kim, Susan K. Hannes, et al. 2019. “Lymphocyte Innateness Is Defined by Underlying Transcriptional States Reflecting a Balance Between Proliferation and Effector Functions”. Nature Communications 10 (1): 687.
Fine-mapping the MHC locus in juvenile idiopathic arthritis (JIA) reveals genetic heterogeneity corresponding to distinct adult inflammatory arthritic diseases.
Hinks, Anne, John Bowes, Joanna E. Cobb, Hannah C. Ainsworth, Miranda C. Marlon, M. E. Comeau, Marc Sudman, et al. 2017. “Fine-Mapping the MHC Locus in Juvenile Idiopathic Arthritis (JIA) Reveals Genetic Heterogeneity Corresponding to Distinct Adult Inflammatory Arthritic Diseases.”. Annals of Rheumatic Diseases 76 (4): 765-72.
Molecular basis for increased susceptibility of Indigenous North Americans to seropositive rheumatoid arthritis.
Scally, Stephen, Soi Cheng Law, Yi Tian Ting, Jurgen Van Heemst, Jeremy Sokolove, Aaron J. Deutsch, E. Bridie Clemens, et al. 2017. “Molecular Basis for Increased Susceptibility of Indigenous North Americans to Seropositive Rheumatoid Arthritis.”. Annals of Rheumatic Diseases 76 (11): 1915-23.
Review: Genetics and the Classification of Arthritis in Adults and Children.
Nigrovic, Peter A., Soumya Raychaudhuri, and Susan D. Thompson. 2018. “Review: Genetics and the Classification of Arthritis in Adults and Children.”. Arthritis Rheumatology 7 (1): 7-17.