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Minokoshi, Yasuhiko, Ronald Kahn, and Barbara Kahn. 2003. “Tissue-Specific Ablation of the GLUT4 Glucose Transporter or the Insulin Receptor Challenges Assumptions about Insulin Action and Glucose Homeostasis”. J Biol Chem 278 (36): 33609-12.

Kulkarni, Rohit, Katrine Almind, Joseph Goren, Jonathon Winnay, Kohjiro Ueki, Terumasa Okada, and Ronald Kahn. 2003. “Impact of Genetic Background on Development of Hyperinsulinemia and Diabetes in Insulin Receptor/Insulin Receptor Substrate-1 Double Heterozygous Mice”. Diabetes 52 (6): 1528-34.

Lee, Zhang, Ivanova, Bonnette, Oesterreich, Rosen, Grimm, et al. 2003. “Developmental and Hormonal Signals Dramatically Alter the Localization and Abundance of Insulin Receptor Substrate Proteins in the Mammary Gland”. Endocrinology 144 (6): 2683-94.

Goldfine, Allison, Clara Bouche, Robert Parker, Caroline Kim, Amy Kerivan, Stuart Soeldner, Blaise Martin, James Warram, and Ronald Kahn. 2003. “Insulin Resistance Is a Poor Predictor of Type 2 Diabetes in Individuals With No Family History of Disease”. Proc Natl Acad Sci U S A 100 (5): 2724-9.

Fisher, Simon, and Ronald Kahn. 2003. “Insulin Signaling Is Required for Insulin’s Direct and Indirect Action on Hepatic Glucose Production”. J Clin Invest 111 (4): 463-8.

Mur, Cecilia, Angela Valverde, Ronald Kahn, and Manuel Benito. 2002. “Increased Insulin Sensitivity in IGF-I Receptor--Deficient Brown Adipocytes”. Diabetes 51 (3): 743-54.

Taniguchi, Cullen, Tatsuya Kondo, Mini Sajan, Ji Luo, Roderick Bronson, Tomoichiro Asano, Robert Farese, Lewis Cantley, and Ronald Kahn. 2006. “Divergent Regulation of Hepatic Glucose and Lipid Metabolism by Phosphoinositide 3-Kinase via Akt and PKClambda/Zeta”. Cell Metab 3 (5): 343-53.

Gunton, Jenny, Rohit Kulkarni, Sun Hee Yim, Terumasa Okada, Wayne Hawthorne, Yu-Hua Tseng, Russell Roberson, et al. 2005. “Loss of ARNT/HIF1beta Mediates Altered Gene Expression and Pancreatic-Islet Dysfunction in Human Type 2 Diabetes”. Cell 122 (3): 337-49.

McGettrick, Aileen, Edward Feener, and Ronald Kahn. 2005. “Human Insulin Receptor Substrate-1 (IRS-1) Polymorphism G972R Causes IRS-1 to Associate With the Insulin Receptor and Inhibit Receptor Autophosphorylation”. J Biol Chem 280 (8): 6441-6.

Kasuga, Fujita-Yamaguchi, Blithe, and Kahn. 1983. “Tyrosine-Specific Protein Kinase Activity Is Associated With the Purified Insulin Receptor”. Proc Natl Acad Sci U S A 80 (8): 2137-41.