Identification of the protein 4.1 binding site to phosphatidylserine vesicles.
Cohen, A M, S C Liu, J Lawler, L Derick, and J Palek. 1988. “Identification of the Protein 4.1 Binding Site to Phosphatidylserine Vesicles.”. Biochemistry 27 (2): 614-9.
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Production, characterization, and use of monoclonal antibodies directed against human blood platelet thrombospondin: immunologic comparison with human endothelial and fibroblast thrombospondins.
Clezardin, P, N R Hunter, J Lawler, J Dawes, and J L McGregor. 1987. “Production, Characterization, and Use of Monoclonal Antibodies Directed Against Human Blood Platelet Thrombospondin: Immunologic Comparison With Human Endothelial and Fibroblast Thrombospondins.”. Seminars in Thrombosis and Hemostasis 13 (3): 261-75.
The structure of human thrombospondin, an adhesive glycoprotein with multiple calcium-binding sites and homologies with several different proteins.
Lawler, J, and R O Hynes. 1986. “The Structure of Human Thrombospondin, an Adhesive Glycoprotein With Multiple Calcium-Binding Sites and Homologies With Several Different Proteins.”. The Journal of Cell Biology 103 (5): 1635-48.
Thrombospondin interactions with fibronectin and fibrinogen. Mutual inhibition in binding.
Lahav, J, J Lawler, and M A Gimbrone. 1984. “Thrombospondin Interactions With Fibronectin and Fibrinogen. Mutual Inhibition in Binding.”. European Journal of Biochemistry 145 (1): 151-6.
A molecular defect of spectrin in a subset of patients with hereditary elliptocytosis. Alterations in the alpha-subunit domain involved in spectrin self-association.
Lawler, J, S C Liu, J Palek, and J Prchal. 1984. “A Molecular Defect of Spectrin in a Subset of Patients With Hereditary Elliptocytosis. Alterations in the Alpha-Subunit Domain Involved in Spectrin Self-Association.”. The Journal of Clinical Investigation 73 (6): 1688-95.
Evidence for calcium-sensitive structure in platelet thrombospondin. Isolation and partial characterization of thrombospondin in the presence of calcium.
Lawler, J, F C Chao, and C M Cohen. 1982. “Evidence for Calcium-Sensitive Structure in Platelet Thrombospondin. Isolation and Partial Characterization of Thrombospondin in the Presence of Calcium.”. The Journal of Biological Chemistry 257 (20): 12257-65.
Addition of an Fc-IgG induces receptor clustering and increases the in vitro efficacy and in vivo anti-tumor properties of the thrombospondin-1 type I repeats (3TSR) in a mouse model of advanced stage ovarian cancer.
Matuszewska, Kathy, Simone Ten Kortenaar, Madison Pereira, Lisa A Santry, Duncan Petrik, Kin-Ming Lo, Byram W Bridle, Sarah K Wootton, Jack Lawler, and Jim Petrik. 2022. “Addition of an Fc-IgG Induces Receptor Clustering and Increases the in Vitro Efficacy and in Vivo Anti-Tumor Properties of the Thrombospondin-1 Type I Repeats (3TSR) in a Mouse Model of Advanced Stage Ovarian Cancer.”. Gynecologic Oncology 164 (1): 154-69.
Endothelial Cell Behavior Is Determined by Receptor Clustering Induced by Thrombospondin-1.
Morandi, Verônica, Jim Petrik, and Jack Lawler. 2021. “Endothelial Cell Behavior Is Determined by Receptor Clustering Induced by Thrombospondin-1.”. Frontiers in Cell and Developmental Biology 9: 664696.
AAV-mediated expression of 3TSR inhibits tumor and metastatic lesion development and extends survival in a murine model of epithelial ovarian carcinoma.
Yu, Darrick L, Ashley A Stegelmeier, Natalie Chow, Amira D Rghei, Kathy Matuszewska, Jack Lawler, Byram W Bridle, James J Petrik, and Sarah K Wootton. 2020. “AAV-Mediated Expression of 3TSR Inhibits Tumor and Metastatic Lesion Development and Extends Survival in a Murine Model of Epithelial Ovarian Carcinoma.”. Cancer Gene Therapy 27 (5): 356-67.
Integration of pro- and anti-angiogenic signals by endothelial cells.
Kazerounian, Shideh, and Jack Lawler. 2018. “Integration of Pro- and Anti-Angiogenic Signals by Endothelial Cells.”. Journal of Cell Communication and Signaling 12 (1): 171-79.