The thrombospondins.
Adams, Josephine C, and Jack Lawler. 2004. “The Thrombospondins.”. The International Journal of Biochemistry & Cell Biology 36 (6): 961-8.
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Thrombospondin-1-induced vascular smooth muscle cell chemotaxis: the role of the type 3 repeat and carboxyl terminal domains.
Lee, Taeseung, Susan M Nesselroth, Eric T Olson, Nowokere Esemuede, Jack Lawler, Bauer E Sumpio, and Vivian Gahtan. 2003. “Thrombospondin-1-Induced Vascular Smooth Muscle Cell Chemotaxis: The Role of the Type 3 Repeat and Carboxyl Terminal Domains.”. Journal of Cellular Biochemistry 89 (3): 500-6.
Thrombospondin-1 as an endogenous inhibitor of angiogenesis and tumor growth.
Lawler, Jack. 2002. “Thrombospondin-1 As an Endogenous Inhibitor of Angiogenesis and Tumor Growth.”. Journal of Cellular and Molecular Medicine 6 (1): 1-12.
Thrombospondin-1 suppresses spontaneous tumor growth and inhibits activation of matrix metalloproteinase-9 and mobilization of vascular endothelial growth factor.
Rodriguez-Manzaneque, J C, T F Lane, M A Ortega, R O Hynes, J Lawler, and M L Iruela-Arispe. 2001. “Thrombospondin-1 Suppresses Spontaneous Tumor Growth and Inhibits Activation of Matrix Metalloproteinase-9 and Mobilization of Vascular Endothelial Growth Factor.”. Proceedings of the National Academy of Sciences of the United States of America 98 (22): 12485-90.
CD36 associates with CD9 and integrins on human blood platelets.
Miao, W M, E Vasile, W S Lane, and J Lawler. 2001. “CD36 Associates With CD9 and Integrins on Human Blood Platelets.”. Blood 97 (6): 1689-96.
Inhibition of angiogenesis by thrombospondin-1 is mediated by 2 independent regions within the type 1 repeats.
Iruela-Arispe, M L, M Lombardo, H C Krutzsch, J Lawler, and D D Roberts. 1999. “Inhibition of Angiogenesis by Thrombospondin-1 Is Mediated by 2 Independent Regions Within the Type 1 Repeats.”. Circulation 100 (13): 1423-31.
Cooperation between thrombospondin-1 type 1 repeat peptides and alpha(v)beta(3) integrin ligands to promote melanoma cell spreading and focal adhesion kinase phosphorylation.
Sipes, J M, H C Krutzsch, J Lawler, and D D Roberts. 1999. “Cooperation Between Thrombospondin-1 Type 1 Repeat Peptides and Alpha(v)beta(3) Integrin Ligands to Promote Melanoma Cell Spreading and Focal Adhesion Kinase Phosphorylation.”. The Journal of Biological Chemistry 274 (32): 22755-62.
Overexpression of thrombospondin-1 decreases angiogenesis and inhibits the growth of human cutaneous squamous cell carcinomas.
Streit, M, P Velasco, L F Brown, M Skobe, L Richard, L Riccardi, J Lawler, and M Detmar. 1999. “Overexpression of Thrombospondin-1 Decreases Angiogenesis and Inhibits the Growth of Human Cutaneous Squamous Cell Carcinomas.”. The American Journal of Pathology 155 (2): 441-52.
Retention of cartilage oligomeric matrix protein (COMP) and cell death in redifferentiated pseudoachondroplasia chondrocytes.
Hecht, J T, D Montufar-Solis, G Decker, J Lawler, K Daniels, and P J Duke. 1998. “Retention of Cartilage Oligomeric Matrix Protein (COMP) and Cell Death in Redifferentiated Pseudoachondroplasia Chondrocytes.”. Matrix Biology : Journal of the International Society for Matrix Biology 17 (8-9): 625-33.
Differential roles of protein kinase C and pertussis toxin-sensitive G-binding proteins in modulation of melanoma cell proliferation and motility by thrombospondin 1.
Guo, N, V S Zabrenetzky, L Chandrasekaran, J M Sipes, J Lawler, H C Krutzsch, and D D Roberts. 1998. “Differential Roles of Protein Kinase C and Pertussis Toxin-Sensitive G-Binding Proteins in Modulation of Melanoma Cell Proliferation and Motility by Thrombospondin 1.”. Cancer Research 58 (14): 3154-62.