Cholesterol promotes autoimmune pathology through T follicular helper cells.

Li, W., & Tsokos, G. C. (2025). Cholesterol promotes autoimmune pathology through T follicular helper cells.. Cellular & Molecular Immunology, 22(8), 951-953.

Publisher's Version

Last updated on 12/05/2025
PubMed

Recent Publications

Aberrant Glycosylation of IgG in Children With Active Lupus Nephritis Alters Podocyte Metabolism and Causes Podocyte Injury.
Aberrant T follicular helper cells generated by TH17 cell plasticity in the gut promote extraintestinal autoimmunity.
Advances in the treatment of systemic lupus erythematosus.
Cholesterol promotes autoimmune pathology through T follicular helper cells.
The ubiquitin E3 ligase TRIM21 suppresses type I interferon signaling via STING degradation and ameliorates systemic autoimmunity.
ADAM9 Promotes Glycolysis in Th17 Cells and Autoimmunity Through Activation of IGF-1 Signaling.

george-tsokos

Twitter

Tsokos lab has focused on the cellular and molecular pathogenesis of systemic lupus erythematosus (SLE). Our laboratory has opened and led the field of molecular abnormalities on immune cells in patients with SLE.

Secondary menu

About
Research
News

Powered byOpenScholar^®Admin Login