Search

Search results

85 results found

IRF4 is a key thermogenic transcriptional partner of PGC-1α.

Publication: Journal Article

Kong, Xingxing, Alexander Banks, Tiemin Liu, Lawrence Kazak, Rajesh R Rao, Paul Cohen, Xun Wang, et al. 2014. “IRF4 Is a Key Thermogenic Transcriptional Partner of PGC-1α.”. Cell 158 (1): 69-83.

Adipocytes fail to maintain cellular identity during obesity due to reduced PPARγ activity and elevated TGFβ-SMAD signaling.

Publication: Journal Article

Roh, Hyun Cheol, Manju Kumari, Solaema Taleb, Danielle Tenen, Christopher Jacobs, Anna Lyubetskaya, Linus T-Y Tsai, and Evan D Rosen. 2020. “Adipocytes Fail to Maintain Cellular Identity During Obesity Due to Reduced PPARγ Activity and Elevated TGFβ-SMAD Signaling.”. Molecular Metabolism 42: 101086.

Interferon regulatory factors are transcriptional regulators of adipogenesis.

Publication: Journal Article

Eguchi, Jun, Qing-Wu Yan, Dustin E Schones, Michael Kamal, Chung-Hsin Hsu, Michael Q Zhang, Gregory E Crawford, and Evan D Rosen. 2008. “Interferon Regulatory Factors Are Transcriptional Regulators of Adipogenesis.”. Cell Metabolism 7 (1): 86-94.

The Synergy between Palmitate and TNF-α for CCL2 Production Is Dependent on the TRIF/IRF3 Pathway: Implications for Metabolic Inflammation.

Publication: Journal Article

Ahmad, Rasheed, Areej Al-Roub, Shihab Kochumon, Nadeem Akther, Reeby Thomas, Manju Kumari, Merin S Koshy, et al. 2018. “The Synergy Between Palmitate and TNF-α for CCL2 Production Is Dependent on the TRIF/IRF3 Pathway: Implications for Metabolic Inflammation.”. Journal of Immunology (Baltimore, Md. : 1950) 200 (10): 3599-3611.

Inflammation causes insulin resistance via interferon regulatory factor 3 (IRF3)-mediated reduction in FAHFA levels

Publication: Unpublished

Yan, Shuai, Anna Santoro, Micah J. Niphakis, Antonio M. Pinto, Christopher L. Jacobs, Rasheed Ahmad, Radu M. Suciu, et al. 2023. “Inflammation Causes Insulin Resistance via Interferon Regulatory Factor 3 (IRF3)-Mediated Reduction in FAHFA Levels”. BioRxiv.

Evan Rosen has received the Roy O. Greep Award for Outstanding Research

News: September 20, 2023

Established in 1944, the Endocrine Society’s Laureate Awards recognize the highest achievements in the endocrinology field, including groundbreaking research and innovations in clinical care. The Roy O. Greep Award for Outstanding Research recognizes meritorious contributions to research in...

Contact

Page

Evan Rosen Lab Division of Endocrinology, Diabetes, and Metabolism Beth Israel Deaconess Medical Center Center for Life Sciences, 7th floor 3 Blackfan Circle Boston, MA 02115 E: erosen@bidmc.harvard.edu Admin: Zain Nichols E: znichols@bidmc.harvard.edu T: 617-735-3322 F: 617-735-3223

Search

Search results

IRF4 is a key thermogenic transcriptional partner of PGC-1α.

Adipocytes fail to maintain cellular identity during obesity due to reduced PPARγ activity and elevated TGFβ-SMAD signaling.

Interferon regulatory factors are transcriptional regulators of adipogenesis.

The Synergy between Palmitate and TNF-α for CCL2 Production Is Dependent on the TRIF/IRF3 Pathway: Implications for Metabolic Inflammation.

Inflammation causes insulin resistance via interferon regulatory factor 3 (IRF3)-mediated reduction in FAHFA levels

Evan Rosen has received the Roy O. Greep Award for Outstanding Research

Contact

Browser for a single cell atlas of human and mouse white adipose tissue

The Heyward family welcomes a new member!

Yangmian Yuan