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Beth Israel Deaconess Medical Center
Alexander S. Banks
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    • Research Area 2: Methods to Study Energy Balance in Mice
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Obesity-Linked PPARγ S273 Phosphorylation Promotes Insulin Resistance through Growth Differentiation Factor 3

Hall, Jessica A., Deepti Ramachandran, Hyun C. Roh, Joanna R. DiSpirito, Thiago Belchior, Peter-James H. Zushin, Colin J Palmer, et al. 2020. “Obesity-Linked PPARγ S273 Phosphorylation Promotes Insulin Resistance through Growth Differentiation Factor 3”. Cell Metabolism.
obesity-linked_pparg_s273_phosphorylation.pdf2.86 MB
Publisher's Version
Last updated on 06/13/2023

Recent Publications

  • Microbial metabolism of mannitol as a tracer for the non-invasive measurement of oral-cecal transit
  • Comparative analysis of naked mole-rat thermogenesis and its potential to maintain euthermia in response to cold
  • A consensus guide to preclinical indirect calorimetry experiments
  • Acute regulation of murine adipose tissue lipolysis and insulin resistance by the TGFβ superfamily protein GDF3
  • Not so fast: Paradoxically increased variability in the glucose tolerance test due to food withdrawal in continuous glucose-monitored mice
  • Analysis of Thermogenesis Experiments with CalR

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Metabolic Research Laboratory at Beth Israel Deaconess Medical Center, Harvard Medical School.

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